Creutzfeldt-Jakob disease with features of obsessive-compulsive disorder and anorexia nervosa: the role of cortical-subcortical systems.

by: OL Lopez, ML Berthier, JT Backer, F Boller

Neuropsychiatry Neuropsychol Behav Neurol, Vol. 10, No. 2. (Apr 1997), pp. 120-4.

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Abstract

The authors examined the clinical and neuropathological characteristics of a patient who developed features of obsessive-compulsive disorder (OCD) and anorexia nervosa (AN) as the initial presentation of Creutzfeldt-Jakob disease. He had mild Parkinsonism and showed deficits in visual scanning, set shifting, graphomotor speed, sequencing, and verbal and nonverbal memory. Neuropathological study showed spongiosis and neuronal loss in cortical (e.g., frontal, temporal, parietal), and especially in subcortical structures (e.g., basal ganglia, thalamus). This study supports the hypothesis of abnormal frontal-striatal functioning in the cause of OCD, even in demented subjects. In addition, the authors discuss the role of frontal-temporal-subcortical dysfunction in the cause of acquired AN.

@article{Lopez1997, abstract = {The authors examined the clinical and neuropathological characteristics of a patient who developed features of obsessive-compulsive disorder (OCD) and anorexia nervosa (AN) as the initial presentation of Creutzfeldt-Jakob disease. He had mild Parkinsonism and showed deficits in visual scanning, set shifting, graphomotor speed, sequencing, and verbal and nonverbal memory. Neuropathological study showed spongiosis and neuronal loss in cortical (e.g., frontal, temporal, parietal), and especially in subcortical structures (e.g., basal ganglia, thalamus). This study supports the hypothesis of abnormal frontal-striatal functioning in the cause of OCD, even in demented subjects. In addition, the authors discuss the role of frontal-temporal-subcortical dysfunction in the cause of acquired AN.}, author = {Lopez, O. L. and Berthier, M. L. and Backer, J. T. and Boller, F. }, citeulike-article-id = {3024930}, journal = {Neuropsychiatry Neuropsychol Behav Neurol}, keywords = {dx, dz, human, initial, of, prion}, month = {Apr}, number = {2}, pages = {120--4}, posted-at = {2008-07-21 17:19:15}, priority = {5}, title = {Creutzfeldt-Jakob disease with features of obsessive-compulsive disorder and anorexia nervosa: the role of cortical-subcortical systems.}, volume = {10}, year = {1997} }

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TY - JOUR ID - Lopez1997 L3 - citeulike-article-id:3024930 TI - Creutzfeldt-Jakob disease with features of obsessive-compulsive disorder and anorexia nervosa: the role of cortical-subcortical systems. JF - Neuropsychiatry Neuropsychol Behav Neurol VL - 10 IS - 2 SP - 120 EP - 4 N2 - The authors examined the clinical and neuropathological characteristics of a patient who developed features of obsessive-compulsive disorder (OCD) and anorexia nervosa (AN) as the initial presentation of Creutzfeldt-Jakob disease. He had mild Parkinsonism and showed deficits in visual scanning, set shifting, graphomotor speed, sequencing, and verbal and nonverbal memory. Neuropathological study showed spongiosis and neuronal loss in cortical (e.g., frontal, temporal, parietal), and especially in subcortical structures (e.g., basal ganglia, thalamus). This study supports the hypothesis of abnormal frontal-striatal functioning in the cause of OCD, even in demented subjects. In addition, the authors discuss the role of frontal-temporal-subcortical dysfunction in the cause of acquired AN. KW - dx KW - dz KW - human KW - initial KW - of KW - prion AU - Lopez, OL AU - Berthier, ML AU - Backer, JT AU - Boller, F PY - 1997/Apr// ER -

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